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Tumour cells induce angiogenesis in the surrounding normal brain. This appears to be necessary for the supply of nutrients and for the recruitment of immune cells to the tumour. Recent studies have shown that astrocytomas form new blood vessels by two different mechanisms, recruitment of endothelial cells from the vasculature and sprouting of preexisting blood vessels. Astrocytomas, as well as glioblastomas, also express a variety of proangiogenic factors. These include vascular endothelial growth factor (VEGF), basic fibroblast growth factor (bFGF), interleukins (IL-1 beta and -8), interferon-gamma and TGF-beta1. Most of these factors have been detected in the peri-tumour area or in the tumour mass itself. However, a role for these factors in the neoangiogenesis of astrocytomas remains unproven. A strong implication of angiogenesis in the malignant process of astrocytic tumours is supported by therapeutic results in experimental models. Tumour regression after treatment with anti-VEGF or anti-angiogenic factors might even result in a more malignant phenotype after


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