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Oct 11, 2010 . Link :

FlexiSign Pro 10.0.1. Multilanguage for Windows 32/64 Bit platform (free, direct link for download)

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Category:Logo designAs a result of the promulgation of the NIH Roadmap we are now in a position to perform large scale functional genomic analyses to assess the role of specific gene products in development and disease processes. We are studying the function of specific signaling pathways in the development of the intestinal epithelium, the epithelial stem cells, the neural crest, and the brain. We have determined that in addition to the canonical Wnt/beta-catenin pathway, Sonic Hedgehog is required to activate the Gli1 gene in both the neural tube and the gut, and that Shh regulates expression of the gut stem cell marker Musashi-1. Based on our recent publication in the literature, we have recently determined that members of the Notch family of receptors are also required for both neural crest induction and stem cell renewal in the developing vertebrate gut, and propose that Notch signaling and Wnt signaling act in parallel in the regulation of stem cell self-renewal. A recent study published in the journal Nature Cell Biology confirms that two proteins required for gastrointestinal stem cell renewal are also required for the renewal of epidermal stem cells. This is an example of how the regenerative properties of the gut may extend beyond the gut into the skin. We have also recently published a study showing that bone morphogenetic protein 4 (BMP4) is required for early anterior patterning of the mouse forebrain, and for the induction of Hox gene expression. Thus, the wide variety of our past and current studies shows that many of the cells that give rise to various organs in the mammalian embryo, and that are defined by their ability to renew, express both Wnt and Shh signaling, and that a large number of the signaling pathways and developmental genes that control these processes are conserved across vertebrate evolution. We are interested in determining how signaling pathways cooperate to activate or repress each other in a tissue-specific manner, and in determining the mechanism of gene activation. We are particularly interested in how signals emanating from the basic helix-loop-helix transcription factor Twist1 coordinate gene expression with specific gene regulatory factors to create a molecular signature that activates a cell to give rise to a specific cell type. We have discovered that Twist1 induces

 

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